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English: Replication and pathogenesis of Equine Influenza Virus (EIV). EIV damages the upper and lower respiratory tract's ciliated epithelial cells thereby causes inability to clear foreign substances. Spike glycoprotein HA fastens to the receptors present on the respiratory epithelial cells and it enters the cells by endocytosis. After endocytosis, EIV undergoes fusion and uncoating. Opening of M2 channel leads to proton entry and subsequent release of viral RNA followed by synthesis of viral structures leading to assembly of EIV. EIV is released from the infected cells by the process of budding.
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Source https://www.frontiersin.org/articles/10.3389/fmicb.2018.01941/full
Author Raj K. Singh, Kuldeep Dhama, Kumaragurubaran Karthik, Rekha Khandia, Ashok Munjal, Sandip K. Khurana, Sandip Chakraborty, Yashpal S. Malik, Nitin Virmani, Rajendra Singh, Bhupendra N. Tripathi, Muhammad Munir, and Johannes H. van der Kolk

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Replication and pathogenesis of Equine Influenza Virus (EIV).

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6 September 2018

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current10:13, 17 November 2020Thumbnail for version as of 10:13, 17 November 2020957 × 720 (604 KB)Guest2625Uploaded a work by Raj K. Singh, Kuldeep Dhama, Kumaragurubaran Karthik, Rekha Khandia, Ashok Munjal, Sandip K. Khurana, Sandip Chakraborty, Yashpal S. Malik, Nitin Virmani, Rajendra Singh, Bhupendra N. Tripathi, Muhammad Munir, and Johannes H. van der Kolk from https://www.frontiersin.org/articles/10.3389/fmicb.2018.01941/full with UploadWizard

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