Lessons
edit| Lessons | Required/Optional | Grade | Done? |
|---|---|---|---|
| Starters | Required | A- | Well done! |
| General Wikimarkup | Required | ||
| Citations | Required | ||
| Article Repair 101 | Required | ||
| Templates | Strongly Recommended | ||
| Infoboxes | Optional | ||
| User Permissions | Optional | ||
| Reviewing articles | Optional | ||
| Advanced Templates | Optional | ||
| Vandalism: Part 1 | Required | ||
| Vandalism: Part 2 | Recommended | ||
| Organizing Articles | Optional |
Types of requirement
edit{Adoptees, ignore this...)
- |bgcolor=19abff|Required
- |bgcolor=3dc63d|Optional
- |bgcolor=dce734|Recommended
Lesson 1: Starters - Answer
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Methylphenidorgil (more commonly reffered to as Gapratron) is a prescription Analgesic and one of the primary drugs used to treat acute cephalalgia (Migraine). While usually taken in pill form it can be administered through a transdermal patch. HistoryeditMethylphenidorgil was discovered in 1968 by Swiss pyshicist Dr. Kim Von Wolfgang as a posible cure to dementia however by the late 70's it was being prescribed as a treatment for acute migraines and in 1981 its patent was bought by Pfizer Incorporated however in 1983 Methylphenidorgil was banned when a link between it and miscarriages was discovered. (see below) ControversyeditIn 1982 the French Mary Anne Frank a migraine sufferer sued Pfizer for 10 million American dollars saying the drug Methylphenidorgil had caused her miscarriage, a inquiry was launched and it was found that Methylphenidorgil caused the body to produce myelin, a hormone that cut of oxygen flow to the foetus. Ms. Frank was paid the sum of 10 million dollars for pschological damage and Methylphenidorgil was subsequently banned in 1982. Another though much less publicised case of controversy was when it was dicovered that researchers had use live animals as test subjects which provoked the outrage of many animal rights organisations including PETA. PharmacologyeditMethylphenidate has binding affinity for both the dopamine transporter and norepinephrine transporter, with the Dextromethylphenidate enantiomers displaying a prominent affinity for the norepinephrine transporter. Both the dextro- and levorotary enantiomers displayed receptor affinity for the serotonergic 5HT1A and 5HT2B subtypes, though direct binding to the serotonin transporter was not observed.  See alsoeditCommentseditYou did a great job. Even though this was not a real drug, you could have fooled me. It is clear you spent time reading over articles to determine proper writing style and formatting. You passed with flying colors, and this is a very high A- grade. Good job! Jock Boy (t/c) Sign 21:01, 30 November 2008 (UTC) | |||||||||||||||||||||||||||||||||||||||||||||||||
