Katefremov
This user is a student editor in Northern_Arizona_University/Writing_in_the_Biological_Sciences_001_(Fall_2019) . |
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editHello, Katefremov, and welcome to Wikipedia! My name is Elysia and I work with the Wiki Education Foundation; I help support students who are editing as part of a class assignment.
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If you have any questions, please don't hesitate to contact me on my talk page. Elysia (Wiki Ed) (talk) 16:43, 6 September 2019 (UTC)
Very Long-Chain Acyl-coenzyme A Dehydrogenase Deficiency (VLCADD) is a recessive fatty-acid metabolism disorder that inhibits and/or prevents the body from oxidizing certain fatty acids [5]. The Acyl-CoA Dehydrogenase Very Long Chain (ACADVL) gene controls the production of very long-chain-acyl-dehydrogenase (VLCAD) and therefore interferes with normal metabolic breakdown of fatty acids [6]. Due to the accumulation of fatty acids in the body, VLCADD can cause fatal symptoms if not treated promptly. Depending on the severity of the disease, signs and symptoms can appear as early as infancy or as late as adulthood and are treated on a personal basis [6]. On average, Very Long-Chain Acyl-coenzyme A Dehydrogenase Deficiency, affects more than 1 in 75,000 newborns [8].
Very Long-Chain Acyl-coenzyme A Dehydrogenase Deficiency (VLCADD) is a recessive fatty-acid metabolism disorder that inhibits and/or prevents the body from oxidizing certain fatty acids [9]. The Acyl-CoA Dehydrogenase Very Long Chain (ACADVL) gene controls the production of very long-chain-acyl-dehydrogenase (VLCAD) and therefore interferes with normal metabolic breakdown of fatty acids [6]. Due to the accumulation of fatty acids in the body, VLCADD can cause fatal symptoms if not treated promptly. Depending on the severity of the disease, signs and symptoms can appear as early as infancy or as late as adulthood and are treated on a personal basis [6]. On average, Very Long-Chain Acyl-coenzyme A Dehydrogenase Deficiency, affects more than 1 in 75,000 newborns [8].